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1.南京铁道医学院附属医院
2.美国犹他大学医学院
*本文系作者在美国犹他大学医学院进修期间完成。
收稿日期:1987-09-11,
纸质出版日期:1989-05
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吴复平, Mohammad S.F., Taylor A.,Jr.. 111In-oxine对人血小板辐射损伤可能性探讨[J]. 核技术, 1989,12(5):309-313.
Fuping Wu, S. F. Mohammad, A. Jr. Taylor. The possibility of radiation impairment of human platelets labelled with 111In-oxine[J]. Nuclear techniques, 1989, 12(5): 309-313.
本文探讨了
111
In-oxine标记人血小板时对血小板辐射损伤的可能性。高放射剂量组和低放射剂量组
111
In-oxine标记的血小板的体外ADP激发聚集功能均正常,且标记的自体血小板静脉注射后,仍能在血循环中维持正常功能。两组结果间无显著性差异。本文结果提示高放射性
111
In-oxine对血小板功能无辐射损伤效应。
Investigation on the possibility of radiation impairment of human platelets labelled with
111
In-oxine has been described. 20 normal volunteers were divided into one high-radioactivity-labelled group (15.5±1.4 MBq) /10
9
platelets
n=10) and one low-radioactivity-labelled group (2.55±0.10 MBq/10
9
platelets
n=10) . The ADP-induced aggregability of
111
In-oxine-labelled platelets in-vitro was normal and the functions of
111
In-oxine-labelled platelets in vivo circulation after reinjection remained unaltered in both groups. No significant difference was found between the two groups. The results indicate that high radioactivity of
111
In-oxine does not impair the platelet function.
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